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Branched-chain amino acid catabolic defect in vascular smooth muscle cells drives thoracic aortic dissection via mTOR hyperactivation

Liming Yu, Tao Huang, Jikai Zhao, Zijun Zhou, Zijun Cao, Yanbang Chi, Shan Meng, Yuting Huang, Yinli Xu, Lin Xia, Hui Jiang, Zongtao Yin, Huishan Wang

2023Free Radical Biology and Medicine26 citationsDOI

Topics & Concepts

PI3K/AKT/mTOR pathwayVascular smooth muscleHyperactivationCatabolismBiologyMechanistic target of rapamycinActivator (genetics)Cell biologyInflammationCancer researchSignal transductionEndocrinologyBiochemistryMetabolismImmunologyReceptorSmooth muscleAortic Disease and Treatment ApproachesAortic aneurysm repair treatmentsATP Synthase and ATPases Research
Branched-chain amino acid catabolic defect in vascular smooth muscle cells drives thoracic aortic dissection via mTOR hyperactivation | Litcius