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LC3 lipidation is essential for TFEB activation during the lysosomal damage response to kidney injury

Shuhei Nakamura, Saki Shigeyama, Satoshi Minami, Takayuki Shima, Shiori Akayama, Tomoki Matsuda, Alessandra Esposito, Gennaro Napolitano, Akiko Kuma, Tomoko Namba‐Hamano, Jun Nakamura, Kenichi Yamamoto, Miwa Sasai, Ayaka Tokumura, Mika Miyamoto, Yukako Oe, Toshiharu Fujita, Seigo Terawaki, Atsushi Takahashi, Maho Hamasaki, Masahiro Yamamoto, Yukinori Okada, Masaaki Komatsu, Takeharu Nagai, Yoshitsugu Takabatake, Haoxing Xu, Yoshitaka Isaka, Andrea Ballabio, Tamotsu Yoshimori

2020Nature Cell Biology229 citationsDOI

Topics & Concepts

TFEBCell biologyAutophagyCell injuryLipid-anchored proteinLysosomeKidneyChemistryRenal injuryBiologyBiochemistryEndocrinologyApoptosisEnzymeAutophagy in Disease and TherapyCalcium signaling and nucleotide metabolismLysosomal Storage Disorders Research
LC3 lipidation is essential for TFEB activation during the lysosomal damage response to kidney injury | Litcius