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DTX2 attenuates Lenvatinib-induced ferroptosis by suppressing docosahexaenoic acid biosynthesis through HSD17B4-dependent peroxisomal β-oxidation in hepatocellular carcinoma

Zhongyan Zhang, Qi Zhou, Zhenchong Li, Fuxin Huang, Ke Mo, Cheng Shen, Xing Niu, Baohua Hou, Chuanzhao Zhang, Shanzhou� Huang, Chuanzhao Zhang, Shanzhou� Huang

2025Drug Resistance Updates15 citationsDOI

Topics & Concepts

Docosahexaenoic acidHepatocellular carcinomaLenvatinibPeroxisomeChemistryCancer researchDownregulation and upregulationCell biologyBiochemistryInternal medicineBiologyReceptorMedicinePolyunsaturated fatty acidFatty acidGeneSorafenibFerroptosis and cancer prognosisImmune cells in cancerCancer, Lipids, and Metabolism
DTX2 attenuates Lenvatinib-induced ferroptosis by suppressing docosahexaenoic acid biosynthesis through HSD17B4-dependent peroxisomal β-oxidation in hepatocellular carcinoma | Litcius