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The hERG channel activator, RPR260243, enhances protective <i>I</i><sub>Kr</sub> current early in the refractory period reducing arrhythmogenicity in zebrafish hearts

Yu Shi, ZhaoKai Pang, Ravichandra Venkateshappa, Marvin Gunawan, Jacob Kemp, Elson Truong, Cherlene Chang, Eric Lin, Sanam Shafaattalab, Shoaib Faizi, Kaveh Rayani, Glen F. Tibbits, Victoria E. Claydon, Tom W. Claydon

2020American Journal of Physiology-Heart and Circulatory Physiology23 citationsDOIOpen Access PDF

Abstract

hERG channel dysfunction causes long QT syndrome and arrhythmia. Activator compounds have been of significant interest due to their therapeutic potential. We used the whole organ zebrafish heart model to demonstrate the antiarrhythmic benefit of the hERG activator, RPR260243. The activator abbreviated APD and increased refractoriness, the combined effect of which rescued induced ventricular arrhythmia. Our findings show that the targeted slowing of hERG channel deactivation and enhancement of protective currents caused by the RPR260243 activator may provide an effective antiarrhythmic approach.

Topics & Concepts

hERGActivator (genetics)Refractory periodZebrafishPharmacologyLong QT syndromeEffective refractory periodMedicineInternal medicineChemistryPotassium channelQT intervalReceptorBiochemistryGeneCardiac electrophysiology and arrhythmiasIon channel regulation and functionECG Monitoring and Analysis
The hERG channel activator, RPR260243, enhances protective <i>I</i><sub>Kr</sub> current early in the refractory period reducing arrhythmogenicity in zebrafish hearts | Litcius