Transforming Growth Factor-β Receptor–Mediated, p38 Mitogen-Activated Protein Kinase–Dependent Signaling Drives Enhanced Myofibroblast Differentiation during Skin Wound Healing in Mice Lacking Hyaluronan Synthases 1 and 3
Yan Wang, Judith A. Mack, Vincent Hascall, Edward V. Maytin
Topics & Concepts
MyofibroblastWound healingCell biologyProtein kinase ATransforming growth factorSignal transductionCancer researchMitogen-activated protein kinaseKinasep38 mitogen-activated protein kinasesBiologyReceptorSkin repairMedicineImmunologyFibrosisPathologyBiochemistryProteoglycans and glycosaminoglycans researchWound Healing and TreatmentsFibroblast Growth Factor Research