Litcius/Paper detail

TAT-RasGAP <sub>317-326</sub> kills cells by targeting inner-leaflet–enriched phospholipids

Marc Serulla, Gabriel Ichim, Filip Stojceski, Gianvito Grasso, Sergii Afonin, Mathieu Heulot, Tim Schober, Robyn Roth, Cédric Godefroy, Pierre‐Emmanuel Milhiet, Kushal Kumar Das, Ana J. García‐Sáez, Andrea Danani, Christian Widmann

2020Proceedings of the National Academy of Sciences30 citationsDOIOpen Access PDF

Abstract

Significance TAT-RasGAP 317–326 can lyse cancer cells in a manner distinct from known programmed cell death pathways through its ability to target specific plasma membrane lipids. The killing properties of this peptide may therefore be difficult for cancer cells to alleviate through resistance-building alterations within known regulated cell death pathways. TAT-RasGAP 317–326 and compounds with similar anticancer activities can potentially complement existing anticancer therapies based on the use of genotoxins or radiation that induce apoptosis.

Topics & Concepts

ApoptosisProgrammed cell deathCancer cellCell biologyBystander effectAlternative complement pathwayBiologyCellCancer researchCancerComplement systemBiochemistryImmunologyImmune systemGeneticsRNA Interference and Gene DeliveryCell death mechanisms and regulationRNA and protein synthesis mechanisms